Posted by Bill
(San Fernando, San Fernando, Philippines) on 05/19/2011 | 1227 Posts
I've been reading alot of research on the thyroid, osteoporosis and diabetes in recent weeks which has led me to some surprising facts and conclusions. Just thought it might help those affected if I broadcast and share some of my thoughts and conclusions.
There are essentially two glands that control formation and destruction of bone in the human body. Calcitonin, a thyroid hormone, controls bone formation in the body and the parathyroid hormone or PTH which controls calcium release and bone destruction. This bone formation/destruction cycle in the body is normal and healthy for the body, provided it is in balance, and is a method of continually renewing old bone.
However if the thyroid is hypo- or insufficient and underperforming, then it is possible that this bone formation/destruction becomes unbalanced -- due to underproduction of calcitonin -- with bone destruction dominating this process and this is when osteoporisis occurs as a problem.
When a healthy amount of Calcitonin is produced from a healthy thyroid, more osteoblasts(bone forming) are produced than osteoclasts(bone destruction) with an accompanying rise in the bone hormone called osteocalcin. Very recent research has revealed that an increase in osteocalcin levels in the blood has a beneficial effect on diabetese. A rise in the body's osteocalcin bone hormone automatically raises the pancreatic production of insulin and also appears to reduce insulin receptor resistance throughout the body. A rise in osteocalcin levels also increases the sexual hormones of the body. Conversely, I have also read, that a possible cause of diabetes is due to the excess calcium in the blood which cause the pancreatic cells -- the Islets of Langerhans -- to calcify internally which greatly reduces production of insulin over time. As an aside, I can't also help wondering if this pancreatic calcification would also affect and reduce production of pancreatic digestive enzymes and bicarbonates as well, which would cause even wider adverse effects on the body's immune system as well.
So, all at once we have made the connection that if we have low levels of thyroid hormone -- caused perhaps by a low iodine or excess fluoride intake -- then this can have a wide domino effect in the body -- leading ultimately to a possible cause of both osteporosis, diabetes as well as a depression of immune system action in the body.
But other problems in this loop relationship can trigger osteoporisis and diabetes for different reasons. For instance, if a person takes the drug Warfarin over a long period for anti-clotting and blood-thinning, this drug is well known to cause osteoporisis -- and so, by logical extension, this drug will also most likely promote diabetes as well.
Another example of this is acid blood, which can also trigger excess calcium release from bone as well as ammonia relase from proteins in muscle tissue to neutralize the acid, all of which leads to loss of weight or a visible wasting of the body.
So, in conclusion, anyone who is suffering from diabetes (type 1 or type 2) would perhaps benefit considerably by also taking nutrients that promote bone formation and bone balance -- such as Iodine or Kelp, Magnesium, Vitamin K2 for example. Also from the research, promoting bone formation and osteocalcin also promotes and helps balance the sexual hormones of the body.
Another different perspective or view on this also explains why taking lugol's iodine for hypothyroid must inevitably help to promote calcitonin and proper bone formation as well as helping diabetes sufferers. This is why both magnesium and iodine have long been considered beneficial for bone formation in the body. However, their beneficial effect on diabetes is a fairly recent research discovery.