Rob (Kentucky) on 08/14/2025

Bronchial allergies are termed “asthma.” The allergens can also be physical agents, such as heat, cold, or sunlight, which cause unusual, strong reactions in hypersensitive individuals.

Form the Book: Healing Factor Vitamin C Against Disease – by Irwin Stone, Linus Pauling, Albert Szent-Gyorgyi 1972, p. 123-124

Source on-line: https://vitamincfoundation.org/stone/

Source download book: https://welib.org/md5/35609ae8abf6e1dcb1cc701374943358

The history of the use of ascorbic acid in the treatment of asthma also dates back to the mid-1930s and is also confusing. It was reviewed in the 1941 paper by Goldsmith (10), who noted the typical pattern of good results of some investigators and of others who failed to confirm these good results. Goldsmith measured the blood ascorbic acid levels of twenty-nine asthmatics and found twenty-two to be below 0.6 mg% (0.7mg% is considered the minimal normal level) and in two of their patients with hay fever only, ascorbic acid was practically absent from their blood (0.07 and 0.08 mg%). On a regime of 300 milligrams of ascorbic acid daily for 1 week, 200 milligrams daily for the second week, and 50 milligrams daily thereafter, six of seven of their asthmatics were unable to maintain blood levels of 1.0 mg%, which was easily achieved by a healthy control group. They interpreted this as a sign that asthmatics had a greater requirement for ascorbic acid. In some of their patients, they found a relationship between the low blood levels of ascorbic acid and the frequency and severity of asthmatic attacks.

Ten years later, in 1951, the literature was again reviewed by Silbert (10). Of the nineteen papers reviewed, thirteen reported benefit, some to complete remission of symptoms, while 6 reported little or no benefit. Silbert suggested that some of these failures may have been due to inadequate dosages of ascorbic acid.

A series of important papers reporting the work of W. Dawson and coworkers (3,11) on the nature of the antagonism of ascorbate on bronchospasm and on the action of ascorbate on smooth muscle, appeared from 1965 to 1967. They showed that spasmogen-induced broncho-constriction in guinea

pigs could be prevented by ascorbic acid. They believed this was due to a direct action of the ascorbate on the bronchial smooth muscle. They also showed that this action is dose-dependent; at low levels it may potentiate the effect of spasmogens, such as histamine, and at higher concentrations it inhibits their spastic effects. This dose-related smooth muscle phenomenon may explain some of the conflicting clinical results of the past four decades.

The protocols for future clinical research using ascorbic acid in asthma should include the megascorbic prophylaxis levels. The dosages would be increased to a point where a therapeutic effect would be obtained. In severe asthmatic attacks, large doses of sodium ascorbate administered intravenously should be tried to relieve the attack. For the safety of this procedure, check the references in Chapter 20 on eye conditions, where doses of 70 grams of sodium ascorbate have been used intravenously without undesirable side effects in the treatment of glaucoma.

Note: Use a good quality Ascorbic acid, made in the USA, third party tested. I choose Nutricost brand, Ascorbic acid Vitamin C because it met all my requirements and it is fairly cheap $20 for 1lb. I am taking one gram (¼ tsp.) powder in half a glass of water 3x/daily. That’s 3,000mg