Morgellons, Hypothyroid & Iodine protocol

Posted By Frances (Cabarlah, Qld.) on 05/20/2015

I am doing Borax/Peroxide external protocol for Morgellons and the Iodine internal protocol for hypothyroid. I am confused now about using peroxide after reading this extract from Dr Dash site re thyroid issues. Should I continue using the peroxide, please?

Quote: Recent advances in research into thyroid cell physiology shows that selenium is very important for thyroid function. There are at least 30 selenium dependent proteins, including the glutathione peroxidase enzyme, and the Iodothyronine deiodinases enzyme (this is the one that converts thyroxine (T4) to bioactive (T3). These proteins all need selenium as a co-factor in order to function properly. The selenoprotein, glutathione peroxidase, protects thyroid cells from damage by hydrogen peroxide (H2O2) produced by the thyroid cell. H2O2 is needed as a normal step in thyroid hormone production, However, too much H202 can damage the thyroid cell. In the event of selenium deficiency, the glutathione peroxidase enzyme cannot do its job protecting the thyroid cell, and the thyroid cells are damaged by excess H2O2. The current theory is that this damaged cell material is then recognized by the immune system as foreign, leading to Hashimoto's autoimmune disease.

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Replied by Frances (Cabarlah, Qld.) on 02/02/2016

Question for Bill or other knowledgeable person: I am hypothyroid, doing iodine protocol, also doing Ted's borax/H2O2 therapy for Morgellons. Is taking H2O2 harming the thyroid? I've read similar info. to this quote before but I don't understand it. Should I stop the H2O2? Advice would be so helpful.

From Bioidentical Hormones 101
By Jeffrey Dach MD

Why is Selenium So Important for Thyroid Function?

The selenoprotein, glutathione peroxidase, protects thyroid cells from damage by hydrogen peroxide (H2O2) produced by the thyroid cell. H2O2 is needed as a normal step in thyroid hormone production, However, too much H202 can damage the thyroid cell. In the event of selenium deficiency, the glutathione peroxidase enzyme cannot do its job protecting the thyroid cell, and the thyroid cells are damaged by excess H2O2. The current theory is that this damaged cell material is then recognized by the immune system as foreign, leading to Hashimoto’s autoimmune disease.

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